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Disordered Actomyosin Contracts in Unexpected Ways
The motion of living cells is in large part due to the interaction of
semi-flexible actin filaments (F-actin) and myosin molecular motors,
which induce the relative sliding of F-actin. It is often assumed that
this simple sliding is sufficient to account for all actomyosin-based
motion. While this is correct in our highly organized striated muscle,
we question the application of this dogma to less ordered actomyosin
systems, thus reexamining a cornerstone of our understanding of cellular
motion.